[OutsiderFan]

What does it all mean?

And how can the higher death rates in the Northeast, Michigan, and Louisiana, be reconciled with the lower death rates on the West Coast? I mean regardless of testing, CA’s death rate is 29th in the U.S.

- Do we understand the implications of reinfections?
- Does original infection destroy T-cells?
- If T-cells are destroyed, are they only in those who develop symptoms, or in everyone exposed?
- Will degree of symptoms with initial infection have any bearing on symptoms with reinfection?
- Can reinfected shed and transmit virus?
- if reinfected can shed and transmit, what does this tell us about vaccine viability?
- What determines who can effectively fight off the virus without symptoms and who can’t?

It seems the data demonstrates there are at least 10x more infections than confirmed cases. Is the population of infected but not confirmed where future confirmed cases mostly come from, or is there another population that develops symptoms and is more rapidly tested, where most new confirmed case growth comes? If the former, then there are a ton of new cases that are going to develop and continue pressuring health care capacity, regardless of NPIs. If the latter, we can assume NPIs are absolutely working to prevent infections in a very significant way.

There was a study of a Boston homeless living facility I saw cited. All 400 something staying there were tested and 36% were positive, all asymptomatic, though one later ended up in hospital. Did they all later test positive, or did some simply not get infected? If not, why not? And how many of the 36% later did or will develop symptoms?

[OutsiderFan]

What does it all mean?

And how can the higher death rates in the Northeast, Michigan, and Louisiana, be reconciled with the lower death rates on the West Coast? I mean regardless of testing, CA’s death rate is 29th in the U.S.

- Do we understand the implications of reinfections?
- Does original infection destroy T-cells?
- If T-cells are destroyed, are they only in those who develop symptoms, or in everyone exposed?
- Will degree of symptoms with initial infection have any bearing on symptoms with reinfection?
- Can reinfected shed and transmit virus?
- if reinfected can shed and transmit, what does this tell us about vaccine viability?
- What determines who can effectively fight off the virus without symptoms and who can’t?

It seems the data demonstrates there are at least 10x more infections than confirmed cases. Is the population of infected but not confirmed where future confirmed cases mostly come from, or is there another population that develops symptoms and is more rapidly tested, where most new confirmed case growth comes? If the former, then there are a ton of new cases that are going to develop and continue pressuring health care capacity, regardless of NPIs. If the latter, we can assume NPIs are absolutely working to prevent infections in a very significant way.

There was a study of a Boston homeless living facility I saw cited. All 400 something staying there were tested and 36% were positive, all asymptomatic, though one later ended up in hospital. Did they all later test positive, or did some simply not get infected? If not, why not? And how many of the 36% later did or will develop symptoms?