[g1313]

You may already know this, but the SCMP is the newspaper of record for Hong Kong, so I would certainly classify it as mainstream media in that is is writing for broad consumption by laymen, as opposed to a medical journal.

Wow, when did the "South China Morning Post" become a mainstream media source in the U.S.?! Agree that this was a little overly alarmist, although the last few paragraphs raise an interesting issue:

But there was one major difference between Sars-CoV-2 and HIV, according to the new study.
HIV can replicate in the T cells and turn them into factories to generate more copies to infect other cells.
But Lu and Jiang did not observe any growth of the coronavirus after it entered the T-cells, suggesting that the virus and T-cells might end up dying together.
The study gives rise to some new questions. For instance, the coronavirus can exist for weeks on some patients without causing any symptoms. How it interacted with the T cells in these patients remained unclear.
Some critically ill patients also experienced cytokine storms, where the immune system overreacts and attacks healthy cells.
But why and how the coronavirus triggers that effect remains poorly understood.

[g1313]

You may already know this, but the SCMP is the newspaper of record for Hong Kong, so I would certainly classify it as mainstream media in that is is writing for broad consumption by laymen, as opposed to a medical journal.

Wow, when did the "South China Morning Post" become a mainstream media source in the U.S.?! Agree that this was a little overly alarmist, although the last few paragraphs raise an interesting issue:

But there was one major difference between Sars-CoV-2 and HIV, according to the new study.
HIV can replicate in the T cells and turn them into factories to generate more copies to infect other cells.
But Lu and Jiang did not observe any growth of the coronavirus after it entered the T-cells, suggesting that the virus and T-cells might end up dying together.
The study gives rise to some new questions. For instance, the coronavirus can exist for weeks on some patients without causing any symptoms. How it interacted with the T cells in these patients remained unclear.
Some critically ill patients also experienced cytokine storms, where the immune system overreacts and attacks healthy cells.
But why and how the coronavirus triggers that effect remains poorly understood.